Antimicrobial Susceptibility StudiesEmergence of plasmid-mediated quinolone resistance genes in clinical isolates of Acinetobacter baumannii and Pseudomonas aeruginosa in Henan, China
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Acknowledgments
This work was supported by the Key Project of Natural Science of Education Department of Henan Province, China (13A610839) and the Science and Technology Innovation Foundation of Xinxiang University (12ZA02). We are grateful to George A. Jacoby for kindly providing the E. coli J53Azr.
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2020, Revista Argentina de MicrobiologiaCitation Excerpt :Although a wide set of plasmids harboring qnrS1 have been described in Enterobacteriales (IncColE, IncFI, IncHI, IncI1, IncL/M, IncN, IncX and IncR), the literature on PMQR in P. aeruginosa is scarce. There are descriptions of aac-(6′)-Ib-cr, qnrA1, qnrD, qnrS1 and qnrVC1; however only the last two markers were associated to β-lactamases, blaTEM-1 and blaVIM-2, respectively3,13. Although studies were carried out to detect PMQR in P. aeruginosa collected since 1990, the recent date of these reports may suggest that there is an ongoing increase in the mobile genetic pool in P. aeruginosa which could provide multiple resistance to the most clinically used antibiotics15.
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